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13 Common Sex-Drive Killers
The Pittsburgh team found that the genes for the "glial cell line derived neurotrophic factor" (GDNF) and the GDNF family ligand, called neurturin, were both effective in treating erectile dysfunction in the rats. The genes were placed in a genetically engineered herpes simplex virus, which was then injected into the rats' damaged cavernous nerve.
Blood hormone studies: Testosterone and/or prolactin levels in the blood may be measured to see if abnormalities in either of these sex hormones are present.
Have more sex. Men who have sex at least once a week are 50 percent less likely to develop erectile dysfunction. Sex improves cardiovascular health, reduces stress, and may lower your risk of prostate cancer.
"The best thing to do is to discuss things outside of the bedroom -- not right after it happens, but days or even weeks later," says sex expert Jennifer Downey, MD, a psychiatrist at New York State Psychiatric Institute and a professor at Columbia University. During this talk, Downey says make certain that your man is aware of the health problems that can be the cause of his ED, and gently suggest he talk to his doctor.
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If the Seat Fits By Charles Downey
Sildenafil has been found to be well tolerated without important side effects. The reported side effects are usually mild, and include headache, flushing, nasal congestion, nausea, dyspepsia, diarrhea, and abnormal vision (seeing a bluish hue or brightness).
Viagra, on the opposite hand, is known to bring effects extraordinarily fast. It's typically enough to require a pill about an hour before the intercourse to see the effects.
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Produces more back pain than other agents; men taking 20 mg report a 6% rate compared with 3% of controls; this mild to moderate pain has been reported to occur bilaterally in the lower lumbar, gluteal, thigh, and thoracolumbar areas; pain tends to be increased in recumbency; anti-inflammatory drugs are generally effective in alleviating this problem and few (0.5%) have discontinued tadalafil because of this problem
At the end of the studies, men who took one of the PDE-5 inhibitors showed improvements on all measures of erectile dysfunction, and the drugs caused few adverse reactions, according to the review.
STUDY SELECTION AND DATA EXTRACTION: All identified research, review articles, and abstracts were assessed for relevance, and all relevant information was included. Priority was given to the primary medical literature and clinical trial reports.
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The cardiac effects associated with sildenafil have been studied extensively. Sildenafil is absolutely contraindicated in patients taking nitrates such as nitroglycerin or isosorbide. This is because sildenafil, like the other PDE-5 inhibitor medications, can potentiate the vasodilatory effects of the nitrate-based medication, resulting in an exaggerated vasodilatory effect that is potentially dangerous. Patients with serious cardiac disease, with exertional angina, or taking multiple antihypertensive medications are advised to seek the advice of a cardiologist before beginning therapy with sildenafil. A number of studies examining the cardiac effects of sildenafil have conclusively shown that no adverse consequences exist under normal circumstances.
There is no set time for how long a man should “last” during sex. But when a man has an orgasm before he wants to, he loses his erection and can’t continue with intercourse. Premature ejaculation can be frustrating and embarrassing. You may feel you don’t have enough time to enjoy sex. You may have difficulty satisfying your partner. For some men, embarrassment about premature ejaculation can cause problems with intimacy and damage their relationships.
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(continued)
Epidemiology of erectile dysfunction
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Fatigue Changes in appetite
This test uses electromagnetic vibration to evaluate sensitivity and nerve function in the glans and shaft of the penis. A decreased perception of vibration may indicate nerve damage in the pelvic area, which can lead to impotence.
Erection begins with sexual stimulation. Sexual stimulation can be tactile (for example, by touching the penis), or mental (for example, by having sexual fantasies). Sexual stimulation generates electrical impulses along the nerves going to the penis and causes the nerves to release nitric oxide, which in turn increases the production of cyclic GMP (cGMP) in the smooth muscle cells of the corpora cavernosa. The cGMP causes the smooth muscles of the corpora cavernosa to relax, and allow rapid blood flow into the penis. The in-coming blood fills the corpora cavernosa, making the penis expand.
The reported frequency of sexual intercourse in this population was between 0.25 – 10 per week (mean 2.4). Evaluation of sexual function log revealed that there was good concordance between the pre-study reported frequency and logged frequency during the study period. Of the 113 men in the study, only 33 (29%) reported having some amount of spontaneous erection, but not suitable for enjoyable intercourse at Visit 1. Following sildenafil therapy, 82/99 (82.8%) men had improved erections (based on GEQ) suitable for intercourse. Seventeen (17.2%) failed treatment. There were no differences in the response rates between the various aetiological groups. A total of 33/99 (33.3%) men reported return of spontaneous erections suitable for penetrative intercourse, at least once, following treatment, of whom 14 (42.4%) did not have any spontaneous erections suitable for intercourse prior to therapy. Of the 74 men reviewed at Visit 4, 59 reported that they would continue using Viagra on a long-term basis (79.7%). Three men had reported complete return of spontaneous erections and had stopped using Viagra (1 at Visit 2 and 2 at Visit 4).
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